hyperoxaluria造句

例句与造句

1. Type II hyperoxaluria is caused by mutations in this gene.
2. Primary hyperoxaluria is a rare autosomal recessive condition which usually presents in childhood.
3. Renal transplant is more effective and this is the primary treatment of severe hyperoxaluria.
4. Hyperoxaluria refers to oxalate derived from dietary sources or that which is secondary to malabsorption.
5. These include tumor lysis syndrome, acute phosphate nephropathy, and occasional cases of enteric hyperoxaluria.
6. It's difficult to find hyperoxaluria in a sentence. 用hyperoxaluria造句挺难的
7. One example is the hereditary disease primary hyperoxaluria, which causes kidney stones at an early age.
8. Primary hyperoxaluria is an autosomal recessive disease, meaning both copies of the gene contain the mutation.
9. This excess glyoxylate is then oxidized by lactate dehydrogenase to produce the oxalate that is characteristic of hyperoxaluria.
10. Hyperoxaluria that can potentially lead to oxalate nephropathy and irreversible renal failure is the most significant abnormality seen on urine chemistry studies.
11. "' Primary hyperoxaluria "'is a rare, inherited condition, resulting in increased excretion of oxalate, with oxalate stones being common.
12. Primary hyperoxaluria is a condition that results in the over production of oxalate which combines with calcium to generate calcium oxalate, the main component of kidney stones.
13. Primary hyperoxaluria, on the other hand, refers to a specific type of hyperoxaluria that is due to a metabolic defect resulting from a heritable genetic defect.
14. Primary hyperoxaluria, on the other hand, refers to a specific type of hyperoxaluria that is due to a metabolic defect resulting from a heritable genetic defect.
15. As noted above ( section on calcium oxalate stones ), people with inflammatory bowel disease ( Crohn's disease, ulcerative colitis ) tend to have hyperoxaluria and form oxalate stones.
16. Perhaps the key difficulty in understanding pathogenesis of primary hyperoxaluria, or more specifically, why AGXT ends up in mitochondria instead of peroxisomes, stems from AGXT's somewhat peculiar evolution.
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